Trigeminal neuralgia - this is pain spreading along the trigeminal nerve or its branches, sometimes with hyper- or hypoesthesia in the zone of its innervation.
What are the causes of inflammation, neuritis, trigeminal neuralgia? Causes of trigeminal neuralgia are various inflammatory (inflammation), traumatic (damage), toxic, infectious (infections, including herpes - postherpetic neuralgia), allergic, infectious-allergic, metabolic effects. Compression of nerves (pinching) in bone, musculoskeletal and osteoarticular canals, prolonged microtrauma, especially in combination with hypothermia, and foci of focal infection also play an important role.
Trigeminal neuralgia clinically manifested by short-term attacks of excruciating pain, often in the area of the 2nd and 3rd branches of the trigeminal nerve. It is characterized by the presence of trigger zones on the skin and mucous membranes. Touching them provokes attacks of pain. In most cases attack of pain accompanied by severe local and general autonomic disorders, the following symptoms: facial hyperemia (redness of the face) and swelling on the affected side, lacrimation, rhinorrhea (discharge of watery mucous discharge from the nose), hypersalivation (salivation, increased secretion of the salivary glands), possibly increased blood pressure ( blood pressure), chills-like trembling, difficulty breathing.
The trigeminal nerve, nervus trigeminus, the 5th pair of human cranial nerves is a mixed nerve that contains sensory, motor and autonomic fibers. The functions of the trigeminal nerve are varied.
Sensitive fibers trigeminal nerves originate from the cells of the trigeminal nerve ganglion, which is called the ganglium trigeminale. It is located in the recess of the pyramid of the temporal bone. The dendrites of these cells form 3 branches and 3 trunks.
1 branch of the trigeminal nerve, first branch(nervus ophthalmicus) - the ophthalmic nerve passes in the lateral wall of the cavernous sinus, later through the superior orbital fissure into the orbit. Then it breaks up into branches, innervates such structures as the outer part of the conjunctiva, the skin of the outer corner of the eye, the upper eyelid, the lacrimal gland, the skin of the scalp to the temporal and parietal regions, the skin of the forehead, the skin of the root of the nose, the cornea, the frontal sinus, the main sinuses , nasal mucosa, nasal skin, posterior cells of the ethmoid bone.
2nd branch of the trigeminal nerve, second branch(nervus maxillaris) - the maxillary nerve passes (exit) through the round foramen and the pterygopalatine fossa. It further breaks down into branches and innervates the following sections: the skin of the temporal region (temporal region, temple), the skin of the zygomatic region (cheekbone), the mucous membrane of the posterior ethmoid cells and the main sinus, the vault of the pharynx (pharynx), the nasal cavity (nose), the soft palate, hard palate, mucous membrane of the tonsils (tonsils), skin of the infraorbital region (infraorbital region), wings of the nose, upper lip, gums of the upper jaw, upper teeth.
3rd branch of the trigeminal nerve, third branch(nervus mandibularis) - the mandibular nerve leaves the skull through the foramen ovale (exit point, place of exit), innervates the following areas: mucous membrane of the cheek, mucous membrane of the lower gum (lower gum), skin of the angle of the mouth (corner of the mouth), skin of the external auditory canal , anterior part of the auricle, temple, all lower teeth, skin and mucous membrane of the lower lip.
Motor fibers The trigeminal nerve originates from the motor nucleus nucleus motorius nervi trigemini. The core is located in the bridge tire. Fibers extending from the nucleus leave the cranial cavity through the foramen ovale. They innervate the masticatory muscles and the anterior belly of the digastric muscle. The axons of the trigeminal ganglion cells form a root and go to the bridge, where they divide into 2 branches.
The descending branch forms the descending spinal tract of the trigeminal nerve, which is responsible for conducting temperature and pain sensitivity. It ends in the nucleus spinalis nervi trigemini. The descending spinal tract and its nuclei are analogous in their function and structure to the posterior horns of the spinal cord. The nuclei and path are divided into 5 segments, as a result of which the innervation of the facial skin in the Zelder zones is located in a ring.
Distinguish primary trigeminal neuralgia(idiopathic, essential, typical) and secondary trigeminal neuralgia(symptomatic trigeminal neuralgia).
With primary neuralgia (mainly of central origin), attacks occur for no reason or are provoked by any movements of the facial muscles.
Secondary neuralgia is usually a complication of the primary disease, has a predominantly peripheral genesis and is often caused by pathological processes in the dentofacial area. The pain is almost constant, periodically intensifying in the form of attacks lasting up to several hours.
Damage to one of the sensory branches of the trigeminal nerve results in a disturbance of all types of sensitivity on the face of a peripheral type in the zone of innervation of this branch. In this case there are symptoms: pain, decreased reflexes, fading reflexes. When the optic nerve is damaged, the conjunctival reflex, corneal reflex, and superciliary reflex are affected. When the motor part of the mandibular nerve is damaged, the mandibular reflex suffers. When the trigeminal nerve ganglion is damaged, all types of sensitivity in the area of 3 of its branches are lost, herpetic eruptions (herpes, herpetic blisters), and trophic disorders are often observed. A lesion of one of the sensitive nuclei in the pons results in a dissociated type of sensitivity disorder - superficial or deep. When the nucleus and oral parts of the spinal tract are damaged, a violation of superficial types of sensitivity in the mouth and nose occurs. If the caudal region is affected, sensitivity is impaired in the area of the outer part of the face. When the optic thalamus and the posterior third of the posterior limb of the internal capsule are affected, contralateral hypersthesia is observed on the face, trunk (body), and limbs (arms, legs) according to the hemitype. When the motor nucleus and its fibers are damaged, peripheral paresis occurs, which is characterized by symptoms such as insufficient muscle tension when chewing, muscle atrophy, retraction in the temple area, angle of the lower jaw, deviation of the lower jaw towards the affected side when opening the mouth. If bilateral peripheral paresis occurs, the lower jaw droops, as a result of which a man or woman cannot chew, cannot close his teeth, or close his mouth. Central paresis of the masticatory muscles on one side does not occur, since the corticonuclear fibers approach the motor nucleus of the trigeminal nerve from both hemispheres of the brain. With bilateral lesions, chewing becomes slightly more difficult (difficulty chewing), and the mandibular reflex is significantly enhanced. Small children have difficulty sucking.
Unfortunately, folk remedies, tablets, drugs, medications, massage, herbs in the treatment of diseases, illnesses, trigeminal neuralgia give a small and short-term effect.
Sarklinik provides comprehensive treatment of trigeminal neuralgia in children, in adults, treatment of inflammation of the trigeminal nerve (nervitis, first, second, third branches) in Saratov, Russia, which includes effective reflexology methods. You can cure trigeminal neuralgia in Saratov!
Sarklinik knows how to treat inflammation of the trigeminal nerve, how to cure neuralgia and trigeminal neuritis ! The following types of neuralgia are treated: neuralgia of the 1st (first) branch of the trigeminal nerve, neuralgia of the 2nd (second) branch of the trigeminal nerve, neuralgia of the 3rd (third) branch of the trigeminal nerve in children and adults. . If you have neuropathy, nerve damage, pain, a cold, the trigeminal nerve hurts on the left, right, you have a cold, a cold, inflammation on the face, paralysis, paresis, you don’t know, how to relieve pain, then contact a doctor at Sarklinik.
There are contraindications. Specialist consultation is required.
Photo: Aniram | Dreamstime.com\Dreamstock.ru. The people depicted in the photo are models, do not suffer from the diseases described and/or all similarities are excluded.
Trigeminal neuralgia does not go away painlessly; it is a rather serious illness. This disease most often affects older women. In some cases, even surgery is required.
The trigeminal nerve originates from the trunk of the anterior part of the pons, located next to the middle cerebellar peduncles. It is formed from two roots - a large sensory root and a small motor one. Both roots from the base are directed to the apex of the temporal bone.
The motor root, together with the third sensory branch, exits through the foramen ovale and further connects with it. In the depression at the level of the upper part of the pyramidal bone there is a semilunar node. Three main sensory branches of the trigeminal nerve emerge from it (see photo).
Neuralgia in translation means pain along the nerve. Having 3 branches, the trigeminal nerve is responsible for the sensitivity of one side of the face and innervates strictly defined areas:
All of them, on their way to the innervated structures, pass through certain openings and channels in the bones of the skull, where they can be subjected to compression or irritation. Neuralgia of the 1st branch of the trigeminal nerve is extremely rare; most often the 2nd and/or 3rd branches are affected.
If one of the branches of the trigeminal nerve is affected, a variety of disorders may occur. For example, the area of innervation may become insensitive. Sometimes, on the contrary, it becomes too sensitive, almost to the point of being painful. Often part of the face seems to sag or becomes less mobile.
Conventionally, all types of trigeminal neuralgia can be divided into primary (true) and secondary neuralgia.
The exact cause of the development of trigeminal neuralgia is not clear; as mentioned above, it is an idiopathic disease. But there are factors that most often lead to the development of this disease.
The reasons for the development of trigeminal neuralgia are varied:
The disease is more typical for middle-aged people, and is more often diagnosed. Females suffer more often than males. Damage to the right trigeminal nerve is most often observed (70% of all cases of the disease). Very rarely, trigeminal neuralgia can be bilateral. The disease is cyclical, that is, periods of exacerbation are followed by periods of remission. Exacerbations are more typical in the autumn-spring period.
So, typical signs of pain syndrome with trigeminal neuralgia:
Among the secondary symptoms of trigeminal neuralgia, phobic syndrome should be distinguished. It is formed against the background of “protective behavior,” when a person avoids certain movements and postures so as not to provoke an exacerbation of the disease.
Symptoms are difficult to interpret correctly if the patient’s pain syndrome is mild.
Due to the fact that all patients suffering from trigeminal neuralgia use only the healthy half of the mouth for chewing, muscle compactions form on the opposite side. With a long course of the disease, dystrophic changes in the masticatory muscles and a decrease in sensitivity on the affected side of the face may develop.
Painful attacks may not be isolated, but follow each other at short intervals. The pathogenesis of the development of trigeminal neuralgia is very diverse:
Other diseases are similar to the symptoms of trigeminal neuralgia. These include temporal tendonitis, Ernest's syndrome and occipital neuralgia. With temporal tendonitis, pain affects the cheek and teeth, headache and pain in the neck.
With occipital neuralgia, pain is usually located in front and behind the head and can sometimes spread to the face.
If the patient has neuralgia, then each attack occurs due to irritation of the trigeminal nerve, due to the existence of trigger, or “trigger” zones. They are localized on the face: in the corners of the nose, eyes, nasolabial folds. With irritation, sometimes extremely weak, they can begin to “generate” a stable, long-lasting painful impulse.
Factors causing pain may be:
Trigeminal neuralgia in an advanced state entails certain consequences:
The risk group consists of older people (usually women), people suffering from cardiovascular diseases or metabolic disorders.
A neurologist needs to differentiate frontal sinusitis, glaucoma, dental diseases, otitis, mumps, ethmoiditis or sinusitis. For this, a comprehensive examination is prescribed.
Typically, the diagnosis of trigeminal neuralgia is made based on the patient’s complaints and examination. Magnetic resonance imaging is important in diagnosing the cause of neuralgia. It allows you to identify a tumor or signs of multiple sclerosis.
Basic diagnostic methods:
If you have been diagnosed with neuralgia, do not be alarmed; in general, the prognosis is favorable, but timely treatment plays an important role.
It is extremely difficult to cure this disease and even radical treatment methods do not always give a positive result. But proper therapy can relieve pain and significantly alleviate human suffering.
The main treatment methods for trigeminal neuralgia include:
Various groups of drugs are used in drug treatment, including:
Before using any drug, consultation with a neurologist is necessary.
Finlepsin for trigeminal neuralgia is one of the most common anticonvulsants. The active ingredient of this drug is carbamazepine. This drug plays the role of an analgesic for idiopathic neuralgia or a disease that occurs against the background of multiple sclerosis.
In patients with trigeminal neuralgia, Finlepsin stops the onset of pain attacks. The effect is noticeable 8 – 72 hours after taking the drug. The dosage is selected only by the doctor individually for each patient.
The dose of Finlepsin (carbamazepine), with which patients can talk and chew painlessly, should remain unchanged for a month, after which it should be gradually reduced. Therapy with this drug can last until the patient notes the absence of attacks for six months.
Other drugs for trigeminal neuralgia:
Each of these drugs has indications for use in trigeminal neuralgia. Sometimes these drugs do not help, so phenytoin is prescribed at a dose of 250 mg. The drug has a cardiodepressive effect, so it should be administered slowly.
Physiotherapeutic procedures include paraffin baths, the use of various types of currents, and acupuncture. To get rid of severe pain, doctors give the patient alcohol-novocaine blockades. This is enough for some time, but the blockades are less and less effective each time.
During surgical treatment, the doctor tries to eliminate the compression of the nerve trunk by the blood vessel. In other cases, the trigeminal nerve itself or its node is destroyed in order to relieve pain.
Surgical treatments for trigeminal neuralgia are often minimally invasive. In addition, the surgical method also includes the so-called. Radiosurgery is a bloodless intervention that does not require any incisions or stitches.
There are the following types of operations:
The type of operation is prescribed depending on the individual characteristics of the patient’s disease.
A characteristic feature of all surgical methods is a more pronounced effect when performed early. Those. The earlier this or that operation is performed, the higher the likelihood of cure.
How to treat neuralgia with folk remedies? When using folk remedies, it is important to remember that only symptoms are relieved in this way. Of course, first of all, you should use folk recipes that can effectively help fight the inflammatory process.
It is important to remember that the use of a particular treatment method should be discussed with a doctor. Pay attention to the consequences that drug treatment may bring.
Folk remedies for treating neuralgia at home:
So, getting rid of ternary neuralgia is possible. You just need to seek help from specialists in time and undergo an examination. The neurologist will immediately prescribe the necessary medications to combat the disease. If such remedies do not help in the fight against trigeminal neuralgia, they resort to the help of a neurosurgeon who solves the problem surgically.
Thank you for the article! I have been suffering from trigeminal neuralgia for a long time. NSAIDs help, but unfortunately relapses are common. Hands down.
Good afternoon everyone, I am writing for those who are desperate and have tried everything due to pain caused by the trigeminal nerve. All the information is on the Internet, I won’t write how I suffered for a whole year and went through everything, including tooth removal, a huge number of paid and free doctors in different fields, including unconventional methods of treatment, I spent a lot of money, nerves and effort to no avail. I understood one thing, and maybe this will help someone, if you haven’t done it yet or have done it, but you were told that this is not the cause of the pain - you need to do an MRI 3.0 of the brain (specifically for NEUROVASCULAR CONFLICT) and with the result, namely with a DISC , go for a consultation not just with a neurologist or neurosurgeon, but with a neurosurgeon who performs operations: MICROVASCULAR DECOMPRESSION OF THE TRIGEMINAL NERVE ROOT.
Because I went to see a neurosurgeon at an expensive clinic with the result, it was written that there was a conflict on the right, degree 1, and on the left, degree 2 (the 3rd strongest), it hurt on the right, i.e. Logically, the left side should also hurt, maybe even more, but it only hurt on the right side, the doctor said that I didn’t need surgery, continue with Finlipsin. But when a month later, quite by accident, I ended up at the Sverdlovsk Regional Oncology Center for a consultation, there they sent me to a neurosurgeon who does just such operations - for decompression, so he looked at the disk and said that the operation was necessary and it would help me, i.e. e. will completely relieve pain. In response to my question about the degrees, he said that on the right (where it hurts) the vein lies on the trigeminal root, but on the left it does not lie, I realized that all these degrees are different.
I agreed to the operation although I had a lot of doubts and it helped, I have not been taking Finlipsin for 2 months, after the operation it hurt a little, but it was different, the pain was tolerable and the headache was healing. I wanted to write this message later to wait a little longer to see what would happen, but I thought that someone was suffering and this information would lead to the right actions.
I am very grateful to the doctor that he saved me from this and made the right decision, and even when I was crying, he invited a patient with the same problem after the operation, he no longer had pain, I finally talked live with a person who had the same thing The same as for me, he (the patient) convinced me that I had to agree. Contact your place of residence where these operations are performed directly, because the same neurosurgeons specialize in certain operations.
I am from Yekaterinburg and had surgery with Pavel Borisovich Gvozdev, he has his own website, I came across it (the website) after the operation, this is for those who are from the Sverdlovsk region, the operation is free, I don’t know how they accept it from other regions. I think other doctors in this specialization will also help you. I will post this message on the forums where I tried to find an answer to my suffering.
Ekaterina, thank you very much. In my current situation, any help and advice is worth its weight in gold.
Trigeminal neuralgia (Trousseau's pain tic, Fothergill's disease, trigeminal neuralgia) is a fairly common disease of the peripheral nervous system, the main symptom of which is paroxysmal, very intense pain in the area of innervation (connection with the central nervous system) of one of the branches of the trigeminal nerve. The trigeminal nerve is a mixed nerve; it provides sensory innervation to the face and motor innervation to the masticatory muscles.
A wide variety of factors underlying the disease, excruciating pain, social and work maladjustment, long-term drug treatment with delayed treatment are not the entire range of reasons that keep this problem at the top of the ranking of neurological diseases. The symptoms of trigeminal neuralgia are quite easily recognizable even by non-professionals, but only a specialist can prescribe treatment. We will talk about this disease in this article.
The trigeminal nerve is the 5th pair of cranial nerves. A person has two trigeminal nerves: left and right; The disease is based on damage to its branches. In total, the trigeminal nerve has 3 main branches: the ophthalmic nerve, the maxillary nerve, the mandibular nerve, each of which breaks up into smaller branches. All of them, on their way to the innervated structures, pass through certain openings and channels in the bones of the skull, where they can be subject to compression or irritation. The main reasons for this can be systematized as follows:
The pathological process can affect both the entire nerve and its individual branches. More often, of course, damage to one branch occurs, but in most cases, untimely treatment leads to progression of the disease and involvement of the entire nerve in the pathological process. There are several stages during the course of the disease. At a late stage (third stage of the disease), the clinical picture changes and the prognosis for recovery significantly worsens. Establishing the cause of the disease in each specific case allows you to most effectively select treatment and, accordingly, speed up healing.
The disease is more typical for middle-aged people, and is more often diagnosed. Females suffer more often than males. Damage to the right trigeminal nerve is most often observed (70% of all cases of the disease). Very rarely, trigeminal neuralgia can be bilateral. The disease is cyclical, that is, periods of exacerbation are followed by periods of remission. Exacerbations are more typical in the autumn-spring period. All manifestations of the disease can be divided into several groups: pain syndrome, motor and reflex disorders, vegetative-trophic symptoms.
Nature of the pain: the pain is paroxysmal and very intense, excruciating, sharp, burning. During an attack, patients often freeze and do not even move; they compare the pain to the passage of an electric current, or a shooting sensation. The duration of the paroxysm is from several seconds to several minutes, but during the day the attacks can be repeated up to 300 (!) times.
Localization of pain: pain can affect both the innervation zone of one of the branches and the entire nerve on one side (right or left). One of the features of the disease is the irradiation (spread) of pain from one branch to another, involving the entire half of the face. The longer the disease exists, the more likely it is to spread to other branches. Localization zones:
Provocation of pain: the development of pain paroxysm can be caused by touching or light pressure on the so-called trigger (trigger) zones. These zones are quite variable in each individual patient. Most often this is the inner corner of the eye, the back of the nose, the eyebrow, the nasolabial fold, the wing of the nose, the chin, the corner of the mouth, the mucous membrane of the cheek or gum. An attack can also be provoked by pressing on the exit points of the branches on the face: the supraorbital, infraorbital, and mental foramen. Pain can also be caused by talking, chewing, laughing, washing your face, shaving, brushing your teeth, applying makeup, even blowing wind.
Behavior during an attack: patients do not cry, do not scream, but freeze, trying not to move, rubbing the pain area.
Motor and reflex disorders:
Vegetative-trophic symptoms: observed at the time of the attack, in the initial stages they are slightly expressed, and as the disease progresses, they are necessarily accompanied by a painful paroxysm:
In the late stage of the disease, a focus of pathological pain activity forms in the visual thalamus (thalamus) in the brain. This leads to a change in the nature and location of pain. Eliminating the cause of the disease in this case no longer leads to recovery. The distinctive features of this stage of the disease are as follows:
The main role in establishing a diagnosis belongs to carefully collected complaints and anamnesis of the disease. During a neurological examination, it is possible to identify areas of decreased or increased sensitivity on the face, as well as changes in the following reflexes:
During the period of remission, a neurological examination may not reveal any pathology. To find the cause of neuralgia, the patient may be shown magnetic resonance imaging (MRI), but it does not always reveal the truth.
The main methods of treating trigeminal neuralgia include:
The main drug used in drug treatment remains carbamazepine (Tegretol). It has been used in the treatment of this disease since 1962. It is used according to a special scheme: the initial dose is mg/day, the dose is gradually increased and adjusted to mg/day in several doses. Once the clinical effect is achieved (cessation of painful attacks), the drug in a maintenance dose is used for a long time to prevent the occurrence of attacks, then the dose is also gradually reduced. Sometimes the patient has to take the drug for 6 months or more. Currently, oxcarbazepine (Trileptal) is also used, which has the same mechanism of action as carbamazepine, but is better tolerated.
In addition to carbamazepine, baclofen 5-10 mg 3 times a day (the drug should also be discontinued gradually) and amitriptyline mg/day are used to relieve pain. Of the new drugs synthesized in recent decades, gabapentin (gabagamma, tebantin) is used. When treating with gabapentin, it is also necessary to titrate the dose until it is clinically effective (the initial dose is usually 300 mg 3 times a day, and the effective dose is mg/day), followed by a stepwise reduction until the drug is discontinued. In order to relieve severe exacerbations, sodium hydroxybutyrate or diazepam can be used intravenously. Complex therapy uses nicotinic acid, trental, cavinton, phenibut, pantogam, glycine, B vitamins (milgamma, neurorubin).
Physiotherapeutic treatment is quite varied. Diadynamic currents, electrophoresis with novocaine, ultraphonophoresis with hydrocortisone, acupuncture, and laser therapy can be used. Physiotherapeutic techniques are used only in combination with drug treatment to achieve a faster and better effect.
In the absence of effect from conservative treatment, as well as in cases where trigeminal neuralgia is caused by compression of the root by an anatomical formation, surgical treatment methods are used:
A characteristic feature of all surgical methods is a more pronounced effect when performed early. Those. The earlier this or that operation is performed, the higher the likelihood of cure. It should also be borne in mind that the disappearance of pain attacks does not occur immediately after surgical treatment, but somewhat remotely (the timing depends on the duration of the disease, the extent of the process and the type of surgical intervention). Therefore, all patients with trigeminal neuralgia need timely consultation with a doctor. Previously, the technique of injecting ethyl alcohol into the nerve branching sites was used. Such treatment often gave a temporary effect and had a high incidence of complications. As the nerve regenerated, the pain returned, so today this method of treatment is practically not used.
Of course, it is not possible to influence all probable causes of the disease (for example, congenital narrowness of the canals cannot be changed). However, many factors in the development of this disease can be prevented:
It should also be taken into account that methods of secondary prevention (i.e. when the disease has already manifested itself once) include high-quality, complete and timely treatment.
Treatment of inflammation of the trigeminal nerve (neuralgia) is carried out using various methods, but is it really possible to do this at home? We will try to answer in detail in today’s material.
This disease is insidious - pain symptoms overtake the victim suddenly and he will have to go a long way to get rid of them.
So what is neuralgia and what are the problems of the disease? The trigeminal nerve is three branched nerves that run along both sides of the face: one of the branches is located above the eyebrows, the other two are on both sides of the nose and in the lower jaw.
Inflammation of this nerve is extremely painful and has a specific nature, the consequences of which are literally visible. When affected, pain appears in the forehead, nose, brow ridges, jaw, neck and chin. Severe attacks of toothache are possible. At the same time, nervous twitching, paleness or redness of the skin, and atrophy of the facial muscles also occur.
The disease occurs for various reasons - it can be independent or a consequence of various infections, overwork and stress. If you notice signs of neuralgia, you should not delay seeing a doctor and begin treatment as quickly as possible.
Since each part of the trigeminal nerve is divided into smaller branches that lead to all areas of the face, the nerve covers it as a whole. These branches are responsible for facial sensitivity.
The first branch is responsible for the eyebrow, eye, upper eyelid and forehead. The second - for the nose, cheek, lower eyelid and upper jaw, the third - for some chewing muscles and the lower jaw.
There are two types of disease:
Doctors are still unable to determine the exact factor why neuralgia occurs, but there are a number of reasons that contribute to the appearance and development of the disease:
To get rid of inflammation, you should take treatment:
The etiology of neuralgia is indeed wide, but it is generally accepted that it usually affects women aged 45 to 70 years. With age, immunity decreases and any physical activity can cause an attack of the disease.
Video: trigeminal neuralgia in the “Live Healthy” program with Elena Malysheva.
Many patients complain of sudden and causeless pain, but also note the occurrence of neuralgia after stressful situations. Doctors are inclined to believe that the inflammation developed earlier - a stressful situation triggered the onset of pain.
The branches of the trigeminal nerve affect motor and sensory fibers, acute pain appears, spasms in the area of the masticatory muscles, all these symptoms indicate inflammation.
Symptoms of facial nerve damage are:
If the disease persists for a long time, paleness or redness of the skin, changes in the secretion of glands, greasy or dry skin, swelling of the face and even loss of eyelashes are possible.
Neuralgia pain is divided into two types:
Typical pain can be triggered by washing, brushing teeth, shaving, applying makeup - actions affecting one of the parts of the face. Pain appears during laughter, smiling and talking, most often occurring after exposure to low temperatures on one of the halves of the facial and ear areas.
The trigeminal nerve is located in the temporal zone, where its three branches are located and pass:
The first two branches have sensitive fibers, the last one has sensory and masticatory fibers, providing active muscular movements of the jaw.
In diagnosing pathology, it is important to consult a doctor in time for an assessment of the pain syndrome and a neurological examination. The diagnosis is based on the patient’s complaints, the specialist determines the type of pain syndrome, its triggers, localization and possible places of damage that cause a pain attack.
To determine the affected area and find out which branch of the trigeminal nerve is damaged, the doctor palpates the patient’s face. Additionally, an examination is carried out for the presence of inflammatory processes in the facial area - sinusitis, sinusitis, frontal sinusitis.
The following instrumental research methods are used:
The disease is difficult to treat, and if painful attacks last more than a day, the patients are placed in the neurological department of the hospital. There, complex therapy is prescribed to prevent the development of the chronic form and relieve acute symptoms.
If the diagnosis is confirmed, then treatment of nerve inflammation begins with eliminating the main pain symptoms. In the future, the causes of the disease are determined (so that the treatment itself is not in vain), tests are prescribed and a full-scale examination of the patient is carried out.
To calm the pain, the patient is prescribed the necessary set of medications and referred to an appointment with an endocrinologist, immunologist, infectious disease specialist and allergist. If one of the specialists discovers a problem, they are prescribed appropriate medications.
Video: inflamed trigeminal nerve - how to identify symptoms and cure?
You should not resort to self-medication for neuralgia without consulting a doctor who will select the necessary drug and its dosage.
You also need to undergo mandatory physiotherapeutic treatment: paraffin-ozokerite, UHF, electrophoresis, magnetic therapy.
Surgical elimination of the cause of neuralgia is used in case of ineffectiveness of drug therapy or when pain persists.
There are two surgical methods:
The first method is trepanation of the posterior part of the cranial fossa. The trigeminal nerve root, which compresses the blood vessels, is separated. A special gasket is placed between the spine and the vessels to prevent compression to prevent relapses.
The radiofrequency destruction method is not so traumatic and is carried out under local anesthesia; current discharges are directed to the affected area, they also destroy the roots of the trigeminal nerve, which are susceptible to pathological processes.
Sometimes one operation is enough, otherwise the effect is repeated several times.
Massage for trigeminal neuritis increases tone and relieves excess muscle tension in certain muscle groups. Blood supply and microcirculation in the inflamed nerve and in the affected superficial tissues improve.
The impact on the reflex zones at the exit points of the branches of the trigeminal nerve of the facial, ear and cervical areas comes first in the massage, after which they work with the muscles and skin.
The massage is performed while sitting, leaning your head back on the headrest to relax the neck muscles. Attention is focused on the sternocleidomastoid muscle, thanks to light massaging movements. Then, with stroking and rubbing movements, they move up to the parotid areas, after which they massage the healthy and affected sides of the face.
The procedure lasts about 15 minutes, on average there are sessions per course of treatment.
The most effective folk remedies and recipes:
Important! We require that even traditional methods be used only under the supervision of a doctor. He will clarify the prescription and, moreover, will tell you whether treatment with such drugs will be effective specifically in your case.
Inflammation of the trigeminal nerve is not fatal, but the consequences are very dangerous.
Video: Fayyad Akhmedovich Farhat (Doctor of Medical Sciences, neurosurgeon of the highest qualification category) about a disease of the facial nerve.
Since a common cause of inflammation of the trigeminal nerve is any disease of the paranasal sinuses (frontal sinusitis, sinusitis) or dental disease, premature therapy will greatly reduce the risk of the problem occurring.
For viral and infectious diseases, anticonvulsants should be taken in parallel with antipyretic and antiviral drugs.
What to do if the trigeminal nerve hurts?
If the pain strikes suddenly, you should immediately consult a neurologist, who will determine the source of pain and methods for eliminating pain syndromes, prescribe the necessary medication or refer you to a neurosurgeon. Before going to the doctor, you can try to temporarily calm the pain using traditional methods of treatment.
Which doctor treats you?
A neurologist deals with the treatment of trigeminal neuralgia, and a neurosurgeon deals with surgical intervention on this basis.
In ICD-10 the disease is coded (G50.0).
Does double vision occur?
Double vision with neuralgia is quite real, often accompanied by hearing loss and noise in one of the ears.
Is it possible to heat inflammation of the trigeminal nerve?
The inflamed area should not be heated, even if relief occurs after this. Heat provokes the progression of inflammation, which can spread to other parts of the face.
Is acupuncture effective?
It is believed that acupuncture is really effective for this disease. It affects certain facial points according to special rules and techniques.
What should a pregnant woman do with this problem?
You need to see a doctor, he will take appropriate measures. Transcutaneous electrical stimulation, electrophoresis with a sanitary agent, and acupuncture during pregnancy are allowed.
In this article I would like to talk about what trigeminal neuralgia is and how to cope with this problem.
At the very beginning, you need to decide on the concepts that will have to be used in this article.
What causes the pain that a person experiences when this nerve is inflamed? This occurs when an artery, nerve, and vein come into contact at the base of the skull, causing irritation. Why can the trigeminal nerve become inflamed? The reasons may be as follows:
By what signs can a diagnosis of “inflammation of the trigeminal nerve” be made? Symptoms of this disease are pain that can appear in any part of the face.
Now it has become extremely clear how pain spreads if a person has inflammation of the trigeminal nerve. The symptoms of this disease can also be confused with the symptoms of other diseases, such as, for example, temporal tendonitis or dental problems. That is why, at the very first symptoms, it is important to seek medical help so that the correct diagnosis is made and proper treatment is prescribed.
Pain in this disease can be of two main types:
It is worth saying that pain alone can make a diagnosis such as trigeminal neuralgia.
How can a correct diagnosis of trigeminal neuralgia be made? Diagnosis of the disease should be carried out exclusively by a doctor. You can make a mistake in diagnosis on your own, and compare the symptoms with a completely different disease. What will the doctor do?
If a patient has trigeminal neuralgia, treatment of this disease can be carried out in various ways. So, it can be conservative, that is, medications and physical therapy can be prescribed. Treatment can also be radical. In this case, minimally invasive procedures are used, as well as surgery.
As mentioned above, if the patient has trigeminal neuralgia, treatment can be conservative. What can the doctor prescribe in this case?
If the patient has an inflamed trigeminal nerve, treatment can be carried out using alcohol blockades. Their main goal: freezing the trigeminal nerve. After this, an analgesic effect occurs. With this treatment, the patient will be injected with the drug “Ethanol” into one of the branches of the trigeminal nerve. Relief occurs almost immediately, pain can disappear for a maximum of a day. However, then she still comes back. If the nerve damage is quite severe, the effect of these injections is not so long-lasting. The number of injections allowed varies depending on the degree of the disease and is prescribed exclusively by the doctor. This treatment also has its disadvantages. This method is fraught with the following complications:
How will the process of alcohol blockade proceed if the patient has inflammation of the trigeminal nerve on the face? Medicines for neuralgia that the doctor may prescribe:
It must be said that this procedure should be carried out exclusively on an outpatient basis, because it requires skills and abilities.
How else can you get rid of such a problem as inflammation of the trigeminal nerve on the face? So, in some cases, the patient may be prescribed surgical intervention. What can the doctor do in this case?
It is worth saying that such operations are minimally invasive.
If the patient has trigeminal neuralgia, treatment can be carried out using radiosurgery such as cyberknife or gamma knife.
There are many advantages of this method of treatment. First of all, this is a non-invasive intervention. This eliminates the risk of bleeding and other complications that may occur during a conventional operation. Also, the patient does not require hospitalization, there is no preoperative preparation. It is also important that anesthesia is not required. And one more huge advantage of this method of treatment: there is no postoperative period. After the procedure, the patient can immediately return to their daily activities.
If the patient has trigeminal neuralgia, treatment can be carried out in the following ways:
If a patient is diagnosed with trigeminal neuralgia, drug treatment is not the only way to get rid of this problem. You can also try to cure yourself with various folk remedies.
Trigeminal neuralgia is a chronic disease that occurs with exacerbations and remissions. A characteristic sign of the disease is attacks of intense shooting pain in the innervation zones of the II, III or, less commonly, I branch of the trigeminal nerve. The concept of “trigeminal neuralgia” also corresponds to: Fothergill’s disease, painful tic, trigeminal neuralgia. Trigeminal neuralgia can be idiopathic or symptomatic (secondary). The basis of treatment for trigeminal neuralgia is the use of anticonvulsants (carbamazepine, phenytoin). Along with this, symptomatic treatment and physiotherapy are carried out.
Trigeminal neuralgia is a chronic disease that occurs with exacerbations and remissions. A characteristic sign of the disease is attacks of intense shooting pain in the innervation zones of the II, III or, less commonly, I branch of the trigeminal nerve. The concept of “trigeminal neuralgia” also corresponds to: Fothergill’s disease, painful tic, trigeminal neuralgia. There are two types of trigeminal neuralgia: primary (idiopathic) and secondary (symptomatic).
Idiopathic trigeminal neuralgia in most cases is caused by compression of the trigeminal nerve root in the area of its entry into the brain stem (as a consequence of a pathologically tortuous loop of the superior cerebellar artery). Therefore, if during a neurosurgical operation a patient is found to have compression of a nerve root by a pathologically tortuous blood vessel, secondary trigeminal neuralgia is diagnosed. However, most patients do not undergo neurosurgical operations and, despite assumptions about the compressive nature of neuralgia, they designate it with the term “primary trigeminal neuralgia,” and the diagnosis “secondary trigeminal neuralgia” is made if pathological processes other than compression.
The pathogenesis of trigeminal neuralgia is traditionally explained in terms of the “pain control gate” theory of Melzack and Wall, which is based on the assertion that fast-conducting, well-myelinated type A fibers and unmyelinated C fibers compete. Neuralgia of the V and IX pairs of the CN are caused by compression of their roots in the area of entry into the brain stem. Demyelination of A fibers is accompanied by the appearance of a large number of additional voltage-dependent sodium channels in the demyelinated areas and the formation of contacts of these areas with type C fibers. As a result of these processes, prolonged and high-amplitude activity of pathologically altered A fibers is formed, which manifests itself in the form of painful paroxysms in the facial area ( oral cavity).
The clinical picture of an attack of trigeminal neuralgia has characteristic signs and therefore diagnosing the disease does not present difficulties for a neurologist. So, typical signs of pain syndrome with trigeminal neuralgia:
In some cases, secondary fascial prosopalgic syndrome may develop. All patients diagnosed with trigeminal neuralgia, both during periods of exacerbation and during remission, use the healthy half of the mouth for chewing, as a result of which muscle compactions are formed in the contralateral part of the mouth (most often the posterior belly of the digastric muscle and the pterygoid muscles). In some cases, auscultation of the temporomandibular joint reveals a characteristic crunch. With a prolonged course of the disease, a dystrophic stage may develop, manifested by moderate atrophy of the masticatory muscles and decreased sensitivity on the affected half of the face.
The clinical manifestations of symptomatic trigeminal neuralgia do not differ from the clinical picture of idiopathic neuralgia, typical signs of which are increasing sensory deficits in the area of innervation of the corresponding branch, the absence of refractory periods and other focal symptoms of damage to the brain stem or adjacent CN (ataxia, nystagmus).
In addition to the above-described forms of neuralgia, neuralgia of individual branches of the trigeminal nerve is distinguished. Nasociliary neuralgia (Charlen's neuralgia) is a rare disease characterized by a stabbing pain radiating to the forehead that occurs when the outer surface of the nostril is touched. Supraorbital neuralgia is as rare a disease as nasociliary neuralgia. A characteristic clinical sign of the disease is paroxysmal or constant pain in the area of the supraorbital notch and the medial part of the forehead (the area of innervation of the supraorbital nerve). Neuralgia of other branches of the trigeminal nerve is possible - infraorbital, lingual, alveolar, buccal nerves, as well as “tic-neuralgia” (a combination of neuralgia of the first branch of the trigeminal nerve and periodic migraine neuralgia).
Postherpetic neuralgia is a disease that occurs as a result of decreased immunity in old age. A characteristic clinical sign is persistent or recurrent facial pain (prosopalgia) that occurs for 3 or more months after the onset of Herpes zoster infection.
The basis of treatment for trigeminal neuralgia is the drug carbamazepine, the dose of which is selected individually for each patient. By reducing the amplitude of potentials in the sensitive nuclei of the spinal and diencephalon, it disrupts the perception of external stimuli (including painful ones). In most cases, 1-2 days after taking the drug, patients feel its analgesic effect, the duration of which, as a rule, is from 3 to 4 hours. When prescribing carbamazepine, you should remember about contraindications to its use (glaucoma, epileptic seizures, bone marrow pathology). In case of ineffectiveness (or low effectiveness) of carbamazepine, another anticonvulsant drug is prescribed - phenytoin, which also has a number of contraindications (kidney disease, liver disease, heart failure).
The dose of carbamazepine, which allows patients to eat and talk painlessly, should remain unchanged for one month, after which it should be gradually reduced. Carbamazepine therapy continues for several months/years and is stopped only if there is complete freedom from attacks for 6 months. Antihistamines (diphenhydramine, promethazine), antispasmodics, and microcirculation correctors (nicotinic acid, pentoxifylline) can enhance the therapeutic effect of carbamazepine. Physiotherapeutic methods for the treatment of trigeminal neuralgia are also used: galvanization with amidopyrine or novocaine, ultraphonophoresis with hydrocortisone and diadynamic currents.
The ineffectiveness of drug treatment is an indication for surgical intervention - microsurgical decompression of the branches of the trigeminal nerve in the area of their exit from the brain stem is performed. However, one should remember the high risk of performing such an operation in elderly patients, as well as in cases where trigeminal neuralgia is accompanied by severe somatic pathology. Recently, the most common treatment for trigeminal neuralgia is the method of percutaneous radiofrequency destruction of the trigeminal nerve roots. Completely new bloodless methods for treating trigeminal neuralgia are also being developed. For example, the so-called “gamma knife” - stereotactic radiosurgical destruction of a sensory root with local gamma radiation.
The optimistic prognosis for trigeminal neuralgia is determined by the cause that caused the development of neuralgia and the age of the patient. Trigeminal neuralgia in a young patient caused by facial trauma is usually easy to treat and does not recur in the future. However, in old age, trigeminal neuralgia, accompanied by metabolic disorders in the body, cannot always be cured.
Often the cause of the development of trigeminal neuralgia is some disease of the teeth or paranasal sinuses (frontal sinusitis, sinusitis). Adequate and timely treatment of these diseases reduces the risk of trigeminal neuralgia. Prevention of exacerbations of trigeminal neuralgia involves minimizing psycho-emotional stress, possible hypothermia, and infectious diseases. For infectious and viral diseases, along with antiviral and antipyretic drugs, it is necessary to take anticonvulsants.
Trigeminal neuralgia is a fairly common disease of the peripheral nervous system, the main symptom of which is paroxysmal, very intense pain in the area of innervation (connection with the central nervous system) of one of the branches of the trigeminal nerve.
Trigeminal neuralgia does not go away painlessly; it is a rather serious illness. This disease most often affects older women. In some cases, even surgery is required.
The trigeminal nerve originates from the trunk of the anterior part of the pons, located next to the middle cerebellar peduncles. It is formed from two roots - a large sensory root and a small motor one. Both roots from the base are directed to the apex of the temporal bone.
The motor root, together with the third sensory branch, exits through the foramen ovale and further connects with it. In the depression at the level of the upper part of the pyramidal bone there is a semilunar node. Three main sensory branches of the trigeminal nerve emerge from it (see photo).
Neuralgia in translation means pain along the nerve. Having 3 branches, the trigeminal nerve is responsible for the sensitivity of one side of the face and innervates strictly defined areas:
All of them, on their way to the innervated structures, pass through certain openings and channels in the bones of the skull, where they can be subjected to compression or irritation. Neuralgia of the 1st branch of the trigeminal nerve is extremely rare; most often the 2nd and/or 3rd branches are affected.
If one of the branches of the trigeminal nerve is affected, a variety of disorders may occur. For example, the area of innervation may become insensitive. Sometimes, on the contrary, it becomes too sensitive, almost to the point of being painful. Often part of the face seems to sag or becomes less mobile.
Conventionally, all types of trigeminal neuralgia can be divided into primary (true) and secondary neuralgia.
The exact cause of the development of trigeminal neuralgia is not clear; as mentioned above, it is an idiopathic disease. But there are factors that most often lead to the development of this disease.
The reasons for the development of trigeminal neuralgia are varied:
The disease is more typical for middle-aged people, and is more often diagnosed. Females suffer more often than males. Damage to the right trigeminal nerve is most often observed (70% of all cases of the disease). Very rarely, trigeminal neuralgia can be bilateral. The disease is cyclical, that is, periods of exacerbation are followed by periods of remission. Exacerbations are more typical in the autumn-spring period.
So, typical signs of pain syndrome with trigeminal neuralgia:
Among the secondary symptoms of trigeminal neuralgia, phobic syndrome should be distinguished. It is formed against the background of “protective behavior,” when a person avoids certain movements and postures so as not to provoke an exacerbation of the disease.
Symptoms are difficult to interpret correctly if the patient’s pain syndrome is mild.
Due to the fact that all patients suffering from trigeminal neuralgia use only the healthy half of the mouth for chewing, muscle compactions form on the opposite side. With a long course of the disease, dystrophic changes in the masticatory muscles and a decrease in sensitivity on the affected side of the face may develop.
Painful attacks may not be isolated, but follow each other at short intervals. The pathogenesis of the development of trigeminal neuralgia is very diverse:
Other diseases are similar to the symptoms of trigeminal neuralgia. These include temporal tendonitis, Ernest's syndrome and occipital neuralgia. With temporal tendonitis, pain affects the cheek and teeth, headache and pain in the neck.
With occipital neuralgia, pain is usually located in front and behind the head and can sometimes spread to the face.
If the patient has neuralgia, then each attack occurs due to irritation of the trigeminal nerve, due to the existence of trigger, or “trigger” zones. They are localized on the face: in the corners of the nose, eyes, nasolabial folds. With irritation, sometimes extremely weak, they can begin to “generate” a stable, long-lasting painful impulse.
Factors causing pain may be:
Trigeminal neuralgia in an advanced state entails certain consequences:
The risk group consists of older people (usually women), people suffering from cardiovascular diseases or metabolic disorders.
A neurologist needs to differentiate frontal sinusitis, glaucoma, dental diseases, otitis, mumps, ethmoiditis or sinusitis. For this, a comprehensive examination is prescribed.
Typically, the diagnosis of trigeminal neuralgia is made based on the patient’s complaints and examination. Magnetic resonance imaging is important in diagnosing the cause of neuralgia. It allows you to identify a tumor or signs of multiple sclerosis.
Basic diagnostic methods:
If you have been diagnosed with neuralgia, do not be alarmed; in general, the prognosis is favorable, but timely treatment plays an important role.
It is extremely difficult to cure this disease and even radical treatment methods do not always give a positive result. But proper therapy can relieve pain and significantly alleviate human suffering.
The main treatment methods for trigeminal neuralgia include:
Various groups of drugs are used in drug treatment, including:
Before using any drug, consultation with a neurologist is necessary.
Finlepsin for trigeminal neuralgia is one of the most common anticonvulsants. The active ingredient of this drug is carbamazepine. This drug plays the role of an analgesic for idiopathic neuralgia or a disease that occurs against the background of multiple sclerosis.
In patients with trigeminal neuralgia, Finlepsin stops the onset of pain attacks. The effect is noticeable 8 – 72 hours after taking the drug. The dosage is selected only by the doctor individually for each patient.
The dose of Finlepsin (carbamazepine), with which patients can talk and chew painlessly, should remain unchanged for a month, after which it should be gradually reduced. Therapy with this drug can last until the patient notes the absence of attacks for six months.
Other drugs for trigeminal neuralgia:
Each of these drugs has indications for use in trigeminal neuralgia. Sometimes these drugs do not help, so phenytoin is prescribed at a dose of 250 mg. The drug has a cardiodepressive effect, so it should be administered slowly.
Physiotherapeutic procedures include paraffin baths, the use of various types of currents, and acupuncture. To get rid of severe pain, doctors give the patient alcohol-novocaine blockades. This is enough for some time, but the blockades are less and less effective each time.
During surgical treatment, the doctor tries to eliminate the compression of the nerve trunk by the blood vessel. In other cases, the trigeminal nerve itself or its node is destroyed in order to relieve pain.
Surgical treatments for trigeminal neuralgia are often minimally invasive. In addition, the surgical method also includes the so-called. Radiosurgery is a bloodless intervention that does not require any incisions or stitches.
There are the following types of operations:
The type of operation is prescribed depending on the individual characteristics of the patient’s disease.
A characteristic feature of all surgical methods is a more pronounced effect when performed early. Those. The earlier this or that operation is performed, the higher the likelihood of cure.
How to treat neuralgia with folk remedies? When using folk remedies, it is important to remember that only symptoms are relieved in this way. Of course, first of all, you should use folk recipes that can effectively help fight the inflammatory process.
It is important to remember that the use of a particular treatment method should be discussed with a doctor. Pay attention to the consequences that drug treatment may bring.
Folk remedies for treating neuralgia at home:
Of course, it is not possible to influence all probable causes of the disease (for example, congenital narrowness of the canals cannot be changed). However, many factors in the development of this disease can be prevented:
It should also be taken into account that methods of secondary prevention (i.e. when the disease has already manifested itself once) include high-quality, complete and timely treatment.
So, getting rid of ternary neuralgia is possible. You just need to seek help from specialists in time and undergo an examination. The neurologist will immediately prescribe the necessary medications to combat the disease. If such remedies do not help in the fight against trigeminal neuralgia, they resort to the help of a neurosurgeon who solves the problem surgically.
Thank you for the article! I have been suffering from trigeminal neuralgia for a long time. NSAIDs help, but unfortunately relapses are common. Hands down.
Good afternoon everyone, I am writing for those who are desperate and have tried everything due to pain caused by the trigeminal nerve. All the information is on the Internet, I won’t write how I suffered for a whole year and went through everything, including tooth removal, a huge number of paid and free doctors in different fields, including unconventional methods of treatment, I spent a lot of money, nerves and effort to no avail. I understood one thing, and maybe this will help someone, if you haven’t done it yet or have done it, but you were told that this is not the cause of the pain - you need to do an MRI 3.0 of the brain (specifically for NEUROVASCULAR CONFLICT) and with the result, namely with a DISC , go for a consultation not just with a neurologist or neurosurgeon, but with a neurosurgeon who performs operations: MICROVASCULAR DECOMPRESSION OF THE TRIGEMINAL NERVE ROOT.
Because I went to see a neurosurgeon at an expensive clinic with the result, it was written that there was a conflict on the right, degree 1, and on the left, degree 2 (the 3rd strongest), it hurt on the right, i.e. Logically, the left side should also hurt, maybe even more, but it only hurt on the right side, the doctor said that I didn’t need surgery, continue with Finlipsin. But when a month later, quite by accident, I ended up at the Sverdlovsk Regional Oncology Center for a consultation, there they sent me to a neurosurgeon who does just such operations - for decompression, so he looked at the disk and said that the operation was necessary and it would help me, i.e. e. will completely relieve pain. In response to my question about the degrees, he said that on the right (where it hurts) the vein lies on the trigeminal root, but on the left it does not lie, I realized that all these degrees are different.
I agreed to the operation although I had a lot of doubts and it helped, I have not been taking Finlipsin for 2 months, after the operation it hurt a little, but it was different, the pain was tolerable and the headache was healing. I wanted to write this message later to wait a little longer to see what would happen, but I thought that someone was suffering and this information would lead to the right actions.
I am very grateful to the doctor that he saved me from this and made the right decision, and even when I was crying, he invited a patient with the same problem after the operation, he no longer had pain, I finally talked live with a person who had the same thing The same as for me, he (the patient) convinced me that I had to agree. Contact your place of residence where these operations are performed directly, because the same neurosurgeons specialize in certain operations.
I am from Yekaterinburg and had surgery with Pavel Borisovich Gvozdev, he has his own website, I came across it (the website) after the operation, this is for those who are from the Sverdlovsk region, the operation is free, I don’t know how they accept it from other regions. I think other doctors in this specialization will also help you. I will post this message on the forums where I tried to find an answer to my suffering.
Ekaterina, thank you very much. In my current situation, any help and advice is worth its weight in gold.
Trigeminal neuralgia is a symptom complex manifested by attacks of excruciating pain localized in the zone of innervation of one or more branches of the trigeminal nerve. This is the most common of all types of neuralgia.
Neurostomatologists distinguish between neuralgia with a predominantly central or peripheral component of pathogenesis (central or peripheral origin). That is, there are forms of diseases whose development is based on a central component, for example, neuralgia due to circulatory disorders in the trigeminal nerve nucleus, or a peripheral component as a consequence of the impact of the pathological process on various parts of the peripheral part of the trigeminal nerve (tumors localized near the trigeminal nerve in the posterior or middle cranial fossa, basal meningitis, diseases of the paranasal sinuses, dental system, malocclusion, facial trauma, etc.). There is no doubt the importance in the origin of trigeminal neuralgia of peripheral origin of the compression (tunnel) factor - compression of the nerve root by pathological formations, more often due to the expansion or dislocation of loop-shaped vessels (usually the superior or anterior inferior cerebellar arteries) with the development, as a rule, of focal demyelination, as well as congenital or acquired narrowing of the infraorbital canal due to thickening of its walls (excess osteogenesis leading to hyperostosis) as a result of local chronic inflammatory processes, most often odontogenic and rhinogenic. Chronic irritation of the branches of the trigeminal nerve is possible with an aneurysm of the vessels of the base of the brain, tumors and cysts of the maxillary sinus, osteoma of the frontal bone, dental diseases, purulent sinusitis, tumors of the trigeminal nerve ganglion. The greatest vulnerability of the first and second branches of the trigeminal nerve (ophthalmic and maxillary nerves) is apparently due to their passage through narrow and long bone canals. A provoking factor can be infection (flu, malaria, syphilis, etc.), hypothermia, intoxication (lead, alcohol, nicotine), etc.
The primary link in pathogenesis is, as a rule, damage to the peripheral segment of the nerve. Under the influence of the compression factor and prolonged subcortical stimulation from the periphery, an algogenic system is formed in the brain, which is stable, highly excitable and responds to any afferent impulses with paroxysmal-type excitation. A unified idea has been created about the mechanisms underlying the disease - this is a multineuronal reflex involving, as a result of long-term pathological impulses from the periphery, both specific and nonspecific structures of the brain stem, subcortical formations and the cerebral cortex.
With trigeminal neuralgia, a complex interaction of organic and functional, peripheral and central changes occurs.
The role of compression of the peripheral branches of the trigeminal nerve has become more clear in the light of the “gate” theory of pain. It has been established that the pain syndrome in this pathology is associated with the selective death of thick myelin fibers, responsible for the rapid conduction of clearly localized pain and the inclusion of a “trigger” of central control - the antinociceptive systems of the brain stem, and the proliferation of thin non-myelin fibers, characterized by the slow conduction of vaguely localized (protopathic) pain. pain. Evidence of the formation of a focus of pathological activity in the central nervous system is increased pain with irritation of the auditory and optic nerves and negative emotions. Clinical manifestations of the disease occur when there is a violation of the relationship between the pain focus and antinociceptive structures at the level of the midbrain and diencephalon, which explains the high incidence of the disease in individuals with vascular pathology of the brain, in which the oral parts of the brain stem are affected relatively often and early.
The debate about whether trigeminal neuralgia is a functional or structural disease has already ended. Experimental and clinical studies have proven that after the onset of the disease, structural changes in the form of swelling, fragmentation and vacuolization are detected in the axial cylinders of the affected branch of the trigeminal nerve, which progress during the course of the disease and in its later stages turn into granular disintegration. The central mechanisms of pain paroxysm are activated secondarily under the influence of pathological afferentation from the periphery.
Thus, the idea of the formation of foci of paroxysmal activity of brain neurons due to irritation of brain structures with a decrease in the threshold of excitation of cortical-subcortical structures under the influence of endocrine metabolic factors, circulatory disorders and immunological changes in trigeminal neuralgia is justified.
Trigeminal neuralgia is predominantly of central origin. The etiology and pathogenesis of trigeminal neuralgia of predominantly central origin have not been fully elucidated. However, apparently, under the influence of endocrine, vascular, metabolic and immunological changes, the reactivity of cortical-subcortical structures (probably nuclear formations of the brain) is disrupted, the excitation threshold of which is significantly reduced. Therefore, any irritation from the periphery, especially irritation of the trigger zones by jaw movement (swallowing, chewing, talking, washing, brushing teeth, touching, laughing, blowing wind, etc.), can cause a reaction from the hypothalamic-stem formations. This leads to the development of painful paroxysms.
Features of symptoms. With trigeminal neuralgia of predominantly central origin, the main clinical picture is a short-term attack (from several seconds to several minutes) of excruciating pain of a very different nature (such as “passage of current”, burning, as well as shooting, tearing, cutting, stabbing) in the innervation zone one or more branches of the trigeminal nerve. The attack stops suddenly, ending abruptly; there is no pain during the interictal period. The area of pain distribution coincides with the zone of innervation of the nerve only conditionally. Usually it goes beyond the boundaries of the innervation of a section of a particular nerve branch. Often spreads vertically, to both cheeks and lower jaw. An attack of pain may be accompanied by reflex contractions of the facial and masticatory muscles in the form of a tonic spasm of the corresponding half of the face. During an attack, patients freeze in a suffering pose, afraid to move. Sometimes they take peculiar poses, fearing that an inadvertent movement will intensify or prolong the paroxysm, they hold their breath or, conversely, breathe heavily. Some patients squeeze the painful area or rub it with their fingers, trying to make movements (smacking) that help quickly stop the pain. Often, during a painful attack, hyperkinesis of the face occurs in the form of clonic twitching of its small muscles, sometimes of all facial muscles (pain tic). Attacks of neuralgia are usually provoked by irritation of trigger (trigger) or algogenic zones, which are a peculiar sign of an altered functional state of the sensitive nuclei of the trigeminal nerve. Trigger zones, found in approximately half of the cases and determined in the zone of innervation of the corresponding branch of the trigeminal nerve, are sometimes migratory in nature. Most often they are located around the mouth and in the gum area, but can be on the skin of the face and in the oral cavity: on the mucous membrane of the cheek, alveolar process, teeth, the mechanical or thermal irritation of which provokes an attack. What matters is the vertical load on the teeth, which occurs when the jaws are sharply clenched, walking on an uneven surface, or suddenly falling from the toes to the heels. The more algogenic zones, the more severe the disease. Their appearance indicates an exacerbation of the disease and, conversely, their disappearance is an indicator of the onset of remission. Sharp pressure on the trigger zone can interrupt an attack of neuralgia.
Painful paroxysms develop mainly in the morning or during the day, rarely at night. As a rule, pain occurs in the area of the second or third branch, sometimes in the area of both branches. Neuralgia of the first branch is extremely rare and one should be very careful when making a diagnosis. Similar symptoms occur with frontal sinusitis, local inflammatory processes, sinus thrombosis, etc. But more often it is the irradiation of pain from the second branch of the trigeminal nerve to the first.
In approximately% of cases, the development of paroxysmal pain is preceded by paresthesia in the form of tingling, “crawling”, as well as constant dull, aching pain in the teeth (one or more), less often in the jaws. Approximately 1/3 of patients undergo various dental procedures in connection with these complaints, including the removal of intact teeth. One of the signs of a relapse of the disease process is the appearance of precursors in the form of a feeling of “grown teeth,” heat, itching, hyperhidrosis, and red spots on the skin of the face.
Trigeminal neuralgia of predominantly central origin is more common in women than in men (ratio 3:2). The disease usually begins between the ages of 40 and 60 years, which suggests the influence of vascular and endocrine metabolic factors on the mechanisms of its development. The course of the disease is chronic, with remissions.
On palpation, pain is detected at the exit points of the trigeminal nerve: foramen supraorbitale, foramen infraorbitale, foramen mentale. In some cases, there are also distant pain points, for example, pain in the middle cervical vertebrae. Trigger zones and hyperalgesia are often detected in the area of the corresponding branch of the trigeminal nerve. In the clinical structure of painful paroxysm, a significant place is occupied by autonomic disorders: hyperemia, swelling of the face, lacrimation, rhinorrhea, hypersalivation, injection of scleral vessels, etc. In rare cases, there may be the opposite phenomena: dryness of the oral mucosa, increased heart rate. During an attack, body temperature may rise by several tenths of a degree on the side of the painful paroxysm. Neurotic disorders are expressed: depressive syndrome, anxiety-phobic, hypochondriacal.
When the Gasserian node is damaged and ganglioneuritis develops, rashes are observed, often in the zone of innervation of the first branch. Trigeminal neuritis is characterized by a sensitivity disorder in the form of hypoesthesia, trophic disorders of the eye (keratitis), reduction or loss of the corneal (if the first branch is affected), mandibular reflexes (if the third branch is affected), weakness and atrophy of the masticatory muscles.
Bilateral trigeminal neuralgia of predominantly central origin. It occurs in women 2 times more often than in men. It occurs mainly between the ages of 40 and 50, and in men - at a younger age.
This is a chronic disease that lasts for decades. As a rule, pain appears on one side, and after some time (a significant time interval - from several months to several years) - on the other. The second and third branches of the trigeminal nerve are most often affected, on one or both sides. Combined damage to these branches is usually observed on one of the affected sides.
Age-related, allergic and vascular factors are important in the pathogenesis. Provoking factors are infections, hypothermia, poor-quality dental prosthetics, mental trauma, etc. Paroxysms of pain usually appear alternately on different sides of the face. Only in some patients is there a simultaneous development of paroxysm on both sides, but still more often with a predominance on any one of them. In 50% of cases, during the acute period of the disease, trigger zones are identified, more often in the nasolabial area, less often in the lateral parts of the skin of the face and in the oral cavity. They are often located symmetrically on both sides, sometimes they are migratory in nature. Most patients experience pain at the exit points on the face of the affected branches of the trigeminal nerve, hypoesthesia with areas of anesthesia, hyperesthesia with areas of hyperpathy (usually in patients in whose treatment destructive methods were previously used), severe autonomic disorders and asthenoneurotic reactions.
Emergency care during an exacerbation of trigeminal neuralgia of predominantly central origin. In case of significantly severe pain syndrome, the administration of analgesics such as tramadol (1-2 ml intravenously slowly or intramuscularly), trabar, tradol, tramagit, tramal, baralgin - 5 ml slowly intravenously, maxigan - 2 and 5 ml intramuscularly. For intractable pain syndrome, diclofenac (syn. dicloran, diclomax, dicloberl, bioran, dik, diclobrew, diclonac, naclofen, revina, olfen, epifenac, feloran) is administered - 75 mg (3 ml) intramuscularly.
The effect of analgesics is enhanced by antihistamines and tranquilizers (diphenhydramine, seduxen, pipolfen), as well as neuroleptics (plegomazine, aminazine), levomepromazine (tizercin). For persistent neuralgia, 2 ml of a 0.25% solution of droperidol is slowly administered intramuscularly or intravenously in combination with the synthetic analgesic fentanyl (2 ml of a 0.005% solution) or a mixture of the following composition: 2 ml of a 50% solution of analgin, 2 ml of a 0.5% solution of novocaine and 1 ml of 2% promedol solution (prepare ex tempore).
At the same time, the anticonvulsant drug carbamazepine (finlepsin, stazein, tegretol, amizepine, mazetol) is prescribed in individually selected doses. If the patient has not previously received this drug, it is prescribed orally, starting with 1 tablet (0.2 g) 1-2 times a day daily, gradually increasing the dose per tablet and bringing it up to 2 tablets (0.4 g) 3-4 times in a day. In% of cases, the clinical effect is achieved on the 2-3rd day of the disease. Patients who have previously received the drug can be immediately prescribed carbamazepine 2-3 tablets (0.4-0.6 g) times a day. It is better to prescribe treatment from a dose that gives a therapeutic effect. After the pain disappears, the dose is gradually reduced to maintenance (to 0.2 - 0.1 g per day).
Trigeminal neuralgia: etiology, pathogenesis, classification, clinical picture, diagnosis, differential diagnosis, treatment.
This is localized pain in the facial area. The disease is has been known for a long time. The mechanism and pathogenesis of this pain syndrome are not clear. Theories of the pathogenesis of trigeminal neuralgia:
1. Mechanical theory
Nerve trunks are compressed at their exit points, namely their oval and round foramina. This theory is justified by the fact that neuralgia is the first■ branches of the trigeminal nerve" is rare, because it exits through the wide superior orbital fissure. And the second and third branches exit from the oval and round foramina, according to researchers (Burdenko), when examining a large number of skulls, it was found that on the right side these holes are much narrower than on the left. And neuralgia occurs on the right much more often than on the left. This theory is supported by the fact that neuralgia is rare at a young age. It occurs after the age of 40, when the growth of bones and the trigeminal nerve root stops throwing over the pyramid of the temporal bone at a right angle, it is stretched due to the fact that the intervertebral discs are reduced and the brain is shifted towards the occipital foramen (Anavekron's theory, 1941).
2. Most authors believe that this disease is associated with inflammatory diseases in the dental system. These are diseases such as: chronic periodontitis* of denticles, calcification of bone canals in which nerve fibers pass to the teeth, retention of 3.5 or 8 teeth, etc.
Of great importance is chronic inflammation of the paranasal sinuses (sinusitis and sinusitis), diseases of the mandibular joint, and cervical osteochondrosis.
Great importance is now attached to the disturbance of vascular tone in hypertension, atherosclerosis, when the trophism of the trigeminal nerve is disrupted.
5. Disruption of connections between the trigeminal nerve and the sympathetic nervous system is of great importance.
6. There are isolated descriptions of the occurrence of neuralgia in two or three generations. But these observations do not give reason to talk about a hereditary factor in the development of trigeminal neuralgia.
Interestingly, during times of severe stress, neuralgia goes away completely. This situation is inexplicable; apparently the stressful situation prevails over the pain syndrome.
Trigeminal neuralgia clinic.
This is a chronic disease characterized by paroxysmal pain.
Pain occurs in the area innervated by any branch of the trigeminal nerve. Right-sided neuralgia is twice as common as left-sided neuralgia. Women get sick more often than men. The disease usually begins after 40 years of age.
A characteristic symptom of this disease is paroxysmal pain that develops suddenly against the background of complete health. Brley of a piercing, cutting nature. Patients describe the pain as follows: as if a nail is being screwed into the face. The pain is intense, unbearable, localized in a certain part of the face. Patients do not touch their faces because this intensifies the attack. The attack lasts up to 1 minute, no more. Between attacks the patient is absolutely healthy, but he does not touch the diseased half of the face, because touching may cause a new attack of pain.
With neuralgia of the first branch, the pain is localized in the area of the eye, superciliary arch, forehead and the anterior surface of the temporal region.
With neuralgia of the second branch, pain is localized in the area of the upper lip, wing of the nose, nasolabial fold, lower eyelid and teeth of the upper jaw, as well as the palate. The patient will ask to remove the diseased tooth, although it is intact.
With neuralgia of the third branch, pain is localized in the lower lip, chin, lower jaw teeth and tongue.
The second symptom of neuralgia is that attacks appear only during the day. During an attack, the patient experiences increased secretion of saliva and nasal secretions, sweat appears on the affected side, and the skin turns red.
If the patient complains of pain and at the same time touches the sore spot or allows the doctor to touch it, then this is not trigeminal neuralgia.
Patients have a trigger zone, which causes a painful attack when touched. Such zones exist in the area of exit of the infraorbital nerve, mental nerve, and supraorbital nerve.
If patients suffer from neuralgia for a long time, then due to nervousness, herpes zoster appears on the skin of the face along the nerve. In addition, graying of hair occurs on the corresponding side. Patients stop eating because when they open their mouth they have a seizure. Patients lose weight quickly. In addition, they do not wash their face, suffer from eczema, stomatitis, tartar deposits, and pustular lesions of the facial skin. Sick men don't shave.
Despite the fact that the neuralgia clinic is very bright, there is still no classification of neuralgia. Our doctors divide neuralgia into:
1. Idiopathic, the cause of which is not clear.
2. Symptomatic (secondary), when there is a cause, for example, chronic sinusitis, after the elimination of which neuralgia remains.
Differential diagnosis of trigeminal neuralgia:
1. Trigeminal neuritis, i.e. nerve inflammation.
The pain is constant, there are almost no intervals between them, and they intensify at night. There are no convulsive phenomena in the facial area with neuritis. The patient does not scream during attacks; he describes the pain as deep, diffuse and dull.
2. Neuralgia of the glossopharyngeal nerve.
This disease was first described in 1910 by Weissburg. The reason is unknown. Characteristic symptoms: severe paroxysmal pain in the root of the tongue, in the velum, in the tonsil, in the ear area. That is, the pain does not spread along the branches of the trigeminal cervix. The pain is unilateral, occurs and intensifies during swallowing, laughing, talking and coughing. During an attack, dry throat appears and
cough.
Trigger area: tonsil and root of tongue.
3. Neuralgia of the pterygopalatine ganglion.
Characterized by severe pain in the upper jaw, palate, root of the nose and eyes. Sometimes the pain syndrome spreads to the neck and upper limb. Patients develop persistent runny nose, lacrimation and photophobia.
4. Glossalgia.
It is characterized by unpleasant sensations in the form of “crawling goose bumps” in half of the tongue. As a rule, there are no painful attacks, the pain is constant. In this case, it is necessary to exclude traumatic moments: sharp edges of teeth, uncorrected dentures.
Glossalgia often accompanies gastritis, pancreatitis, and cholecystitis. Prosthetics with different metals lead to the emergence of electrical potentials that cause a burning sensation in the tongue.
5. Osteochondrosis of the cervical spine.
The pain is constant, intensifies when turning the head. It is necessary to take an x-ray of the cervical spine and exclude osteochondrosis. - .
6. Arthritis of the temporomandibular joint. There are no paroxysmal pains.
7. Pulpitis.
Characterized by pain in the tooth, which intensifies at night. A carious tooth is visible in the oral cavity. With pulpitis, the pain is prolonged, and with neuralgia it lasts up to 1 minute.
8. Sinusitis.
Often, after eliminating pulpitis, neuralgia remains, because nerve toxicity occurred
9. Malignant tumors of the upper or lower jaw.
The patient will complain of pain in the teeth, although they may be intact. The pain is constant, not paroxysmal.
10. Inflammation of the middle ear.
The pain is constant. There will definitely be an elevated body temperature, which is not the case with neuralgia.
Treatment of trigeminal neuralgia.
Because The cause of this disease is not clear, the treatment is complex, mainly symptomatic. After treatment, the pain may return.
1. Blockade with a 1% novocaine solution at the exit points of the branches of the trigeminal nerve (supraorbital, infraorbital, mental nerves). A 1% solution is used, rather than 2%, which is more often used in the clinic, because Vishnevsky proved that small concentrations of novocaine have a positive effect on tissue metabolism "and have a therapeutic effect due to nerve blockade. And a 2% solution with prolonged use causes nerve degeneration, i.e. neuralgia turns into trigeminal neuritis. 1 g can be administered simultaneously dry matter of novocaine.
The blockade is performed 2 times a week, 5-10 ml of a 1% solution. After administration, tissue swelling occurs, so the blockade is not performed often so that swelling due to mechanical compression of the nerve does not aggravate the process. It is necessary to conduct 10-12 sessions, which last on average for 2 months.
2. Acupuncture. This method is based on the connection of internal organs with facial skin. This technique is scientifically substantiated and should be carried out by a specialist who has completed special courses.
3. Tissue therapy. Academician Filatov proposed this method in 1933 with the aim of stimulating the body's immune defense. Vegetable or animal protein is taken, sterilized and injected subcutaneously, usually into. abdominal or chest wall, but not in the area of the face. Long-term absorption of this protein stimulates the immune system. You can prescribe aloe and vitreous extract.
4. Oxygen therapy. This method is used in the facial area (20-50 ml of oxygen is injected subcutaneously) or a pressure chamber is used (3 atm for an hour).
5. Vitamin B]2, - administered intramuscularly: Use no more than 8-10
injections.
6. Physiotherapy uses diadynamic currents; they provide an analgesic effect, increase the threshold of pain sensitivity, and normalize vegetative processes. 7. Anticonvulsants are used as medications: finilin (dielintin) and finlepsin - 1 tablet 3 times a day. There are other treatment methods that our department views negatively:
1. Use of boiling water. This method was proposed by the American doctor Yeager. He introduced boiling water to the site where the branches of the trigeminal nerve exit the skull (oval or round foramen). Here, 10 years ago, this method was widely used by Livshits in Saratov. The essence of the method is that soft tissue is burned at the base of the skull. After scarring of the burn, the tissue compresses the nerve and no other conservative methods help.
2. Alcoholization with 80% alcohol. When alcohol is injected into a nerve, intravital degeneration of the nerve fiber occurs. The pain returns after 8-10 months, and other treatment is no longer effective.
3. The method was proposed by Professor Kurbangaliev at our institute. This is a surgical treatment method: transection of the trigeminal nerve root or removal of the Gasserian ganglion. The operation is very difficult and has many complications: patients lose memory and have poor orientation in the external environment. Only three such operations were performed; now they are not recommended due to severe complications; headaches that occur as a result of loss of cerebrospinal fluid and air entry into the subarachnoid space, imbalance with a tendency to fall to the affected side, atoxicity in the arm and leg on the side of the operation, serous meningitis, Borner's syndrome, nystagmus.
The hardest thing is death on the operating table.
The trigeminal nerve belongs to the 5th pair of cranial nerves and has branches - the ophthalmic one, located in the upper jaw and located in the lower jaw. When the inflammatory process develops in the area of this nerve, they speak of neuralgia.
What it is?
Trigeminal neuralgia is a chronic disease in which the branches of the nerve are affected, as a result of which the patient suffers constant paroxysmal pain in the area of their innervation. With this pathology, pain occurs more often in one half of the face.
Common causes of facial trigeminal neuralgia are:
People at risk are:
The main clinical symptom of trigeminal neuralgia is pain, usually localized on one half of the face. The attack occurs suddenly, with the slightest irritation of the affected nerve. The patient complains of shooting pain, which is often equated to electric shocks.
The pain does not last long, usually no more than a few minutes. After this, a period of remission begins, which can last up to several weeks or months, but as the disease progresses, pain occurs more often, and the intervals between them become shorter.
With neuralgia of the 1st branch of the trigeminal nerve, pain is localized in the area of the wing of the nose, eye, eyebrow, temple, crown.
The next attack of pain is provoked by simple actions of the patient:
In addition to pain, before effective treatment begins, symptoms of trigeminal neuralgia include the following conditions:
Diagnosis of trigeminal neuralgia begins with a visit to a neurologist. The doctor examines the patient during remission and after influencing pain points, the influence of which can provoke an attack of pain, collects an anamnesis of life and illness, and prescribes additional studies:
Treatment of the disease is carried out comprehensively; the main steps in the treatment of trigeminal neuralgia are as follows:
To relieve an attack of trigeminal neuralgia and prevent pain in the future, the patient is prescribed the drug Finlepsin. This medicine belongs to the group of anticonvulsants and helps reduce the excitability of nerve fibers and inhibit the production of neurotransmitters.
This drug can be taken strictly according to the doctor’s indications and in an individually designated dosage, since the tablets have a number of serious contraindications.
In addition to Finlepsin, the patient is prescribed:
During stable remission, physiotherapeutic and sanatorium treatment is indicated.
Surgical treatment of trigeminal neuralgia
If conservative treatment methods are not effective, the patient is prescribed surgery. The main indications are brain tumors, the presence of narrowed areas in the nerve exit channel from the skull, pinched nerve branches, etc.
In modern surgery, surgery to eliminate trigeminal neuralgia is performed with a laser. This intervention is minimally invasive and is well tolerated by the patient.
If you seek medical help on time and receive proper treatment, the prognosis for the disease is generally favorable. Following simple preventive recommendations allows you to achieve stable remission or completely get rid of the problem.